Trigger Point Therapy

Trigger Point Therapybe logical to assume that something must irritate
the terminal parts of sensory and motor neurons.
IntroductionThis something is a tension in the skeletal muscles,
Medical massage therapy procedure consists ofincluding trigger points that are not associated with
mobilization of skin, fascia and muscular tissue,motor trigger points (since they are located in
trigger point therapy, and post-isometric relaxationother parts of the skeletal muscle). Keep in mind
techniques. Each of these modalities is equallythat any inflammatory condition, whether in motor
important in order to reach rapid and sustainedend plates or in muscular tissue, means that there
results. For decades, massive utilization of medicalis a decreased amount of blood supply to this
massage has proven to be a safe and veryinflamed tissue. From this it follows that gradual
effective method of treatment for the supportischemic compression can be viewed as an
and movement system disorders, inner organanti-inflammatory effort.
disorders, stress management, and more.There is no doubt that myofascial pain can be the
In the last few years, there have been numerousresult of peripheral nerve abnormalities. An
arguments in within the professional communityexample of this would be the irritation of the
about practitioners utilizing manual therapy andsciatic nerve by an over-tensed piriformis muscle
trigger point therapy. In recent professionalresulting in the formation of trigger points in
publications many authors have been raising themuscles innervated by the sciatic nerve. This list
following questions: Is a trigger point a formationcan be continued because any peripheral nerve's
of fibroconnective tissue in muscles? Haveentrapment in the key areas will cause formation
histological studies ever been done on triggerof trigger points in muscles innervated by this
points? Is there a theory of peripheral nerve painnerve.
at the motor end plate a new theory and theIf one examines a patient with peripheral arterial
only theory? Are ischemic compression techniquesdisorder (e.g., Buerger’s disease) one will find
for trigger point therapy safe and effective?numerous active and dormant trigger points in the
The brief answers on aforementioned questionsleg and foot muscles. It would most certainly be
are:agreeable that insufficient arterial blood supply as
1. Fibroconnective tissue formation in muscles isa result vascular abnormality is responsible for the
myogelosis, an incurable muscular pathology.formation of trigger points in the skeletal muscles
2. In many cases myogelosis is the result ofrather than abnormalities in the motor end-plates.
inadequate treatment of trigger points.The same is true for trigger points in the skeletal
3. A trigger point is a pinpoint localization of painmuscles, which are developed as a result of
that can be found in muscles, connective tissue,chronic visceral disorders (e.g., patients with
and periosteum. The morphology of this point ofcardiac disorders exhibit active trigger points in
pain is such that the demand of blood supply isthe trapezius, levator scapulae, and rhomboideus
much higher than the actual blood supply.muscles). In such cases the end-plate
4. The theory of peripheral nerve pain at theabnormalities do not have anything to do with
motor end plate is not a new theory.formation of trigger points in the skeletal muscles.
5. Any theory must be supported by clinicalThey are the result of the phenomenon of
output.convergence of pain stimuli within the same
6. Ischemic compression as a method of triggersegments of the spinal cord, which are responsible
point therapy has been proven by at least 4for the innervation of both the affected inner
decades of massive utilization as a safe andorgan and the skeletal muscles. In 1955 Dr. Glezer
effective method.and Dalicho formulated the theory that still stands
7. Ischemic compression techniques are applied byclinically proven. They proposed and developed
gradually increasing pressure, thus excluding themaps of reflex zone abnormalities of the skin,
possibility of doing harm to the patient and to thefascia and muscles, including trigger point
therapist.development.
In the search for true understanding ofThe Energy Crisis Theory
pathophysiology, the body’s sophistication andThere is another theory, which links formation of
complexity requires us to take an integrativetrigger points with the shortage of ATP in the
approach to any issue. Thus I would like toaffected muscles as a result of insufficient arterial
present to the reader a short scientific review ofcirculation. ATP is the energy source for cellular
the trigger point issue and the trigger pointfunction, including muscles. Authors of this theory,
therapy concept.called the Energy Crisis Theory, pointed out the
If you would like to view hands on demo, pleaseformation of the trigger points in very healthy
click the link below:athletes who did not have signs of peripheral
The Nature of Trigger Pointsnerve abnormalities and still developed active
There is no statement in the modern scientifictrigger points. Gradual increase of the resting
literature that calls a trigger point a "taut band ofmuscular tone in normal muscles triggers local
fibro-connective tissue." However, it was oncevasoconstriction, interstitial edema, and ATP
used in the late 19th/early 20th century untilexhaustion with the subsequent formation of
histological studies conducted by German scientistsactive trigger points. Prof. D. Simons reviewed this
(Glogowski, and Wallraff, 1951; Miehlke et al., 1950)theory as well, and even used extensively works
showed that there is no connective tissuedone by his colleagues, Dr. D.R Hubbard and Dr.
proliferation (myogelosis) in the area of a triggerG.M. Berkoff, in his own research.
point in muscles. "In our opinion, fibrositis (in regardTrigger Point Therapy Protocol
to trigger points) has become a hopelesslyUltimately trigger point therapy has the following
ambiguous diagnosis... is best avoided" (Travell,goals:
Simons, 1983). However, connective tissue will1. Eliminate protective muscular tension in the
grow between muscle fibers when a core of themuscles that harbor active trigger points.
myogelosis is formed (Glogowski, and Wallraff,2. Eliminate condition of the hyperirritability of the
1951). Myogelosis is a clinical outcome of years ofperipheral receptors, especially pain receptors.
reactivation of the active trigger point in the3. Block the pain-analyzing system of the patient.
same area. At the same time, trigger point4. Produce reflex vasodilation.
therapy is useless if the core of the myogelosis is5. Eliminate local ischemia.
already formed.To effectively achieve these goals the practitioner
In 1843, for the first time, the German physicianshould conduct trigger point therapy utilizing
Dr. F. Froriep described trigger points as painfulseveral equally important components:
formation in skeletal muscles. In 1921 another1. Detect location of the active trigger point.
German scientist, Dr. H. Schade, examined them2. Detect the pathway of pain radiation and
histologically and formed the concept ofexamine tissues along this pathway in case
myogelosis. In 1923 the British physician Dr. J.satellite trigger points are formed.
Mackenzie offered the first pathophysiological3. Place finger in the trigger point. Slowly apply
explanation of the trigger point formationvertical compression of the tissues until the
mechanism and formulated the concept of thepatient feels the first sign of pain. As soon as he
reflex zones in the skeletal muscles where theor she reports it stop increasing pressure but
central and peripheral nervous system play amaintain it at that same level. After 10 seconds of
critical role. The reflex zones concept was furtherpressure application, the pain that the patient
developed by the American scientist Prof. I. Korrinitially felt will disappear. The patient should
in 1941 in a series of brilliantly designedimmediately report to you as soon as he or she
experimental studies. Thus, the trigger pointfeels the pain cease. During the next 20 seconds
concept was developed long before the work ofthe practitioner will be able to get to the "bottom"
Travell and Simons, who based their publicationof the trigger point without unwanted activation
(see references in "Trigger Point Manual" byof the pain analyzing system and generating
Travell and Simons) on the works of theprotective muscular tension in the affected
scientists mentioned.muscle or muscles in the region.
There are numerous published results of4. To accomplish the first three goals apply
histological evaluations of the trigger point areas.effleurage and kneading techniques on the
Even in the short list of references at the end ofaffected muscles in the inhibitory regime for 5-7
this article you can find ample evidence underminutes (comfortable gradual increase of
references 5, 6, 7, 13, and 15.pressure, in the same direction of the strokes).
It is misleading to state that Dr. Travell and Dr.5. Exit the trigger point as fast as possible to
Simons recommended using ischemic compressionproduce quick and effective vasodilation and
for trigger point therapy. They advocatedelimination of the local ischemia.
injection, stretch and spray techniques, andThe correct protocol of trigger point therapy
muscle energy techniques for trigger pointdoes not have pitfalls. This protocol is equally
therapy. Although, Travell and Simons did mentioneffective for the motor trigger points, as well as
ischemic compression as an option based on thefor other trigger points. The applied pressure is
European medical sources, they nevernever strong enough to go over the patient's
recommended it as a treatment method.threshold of pain, causing the pain and injury of
The Role of Vasodilators in Local Ischemiamotor nerve endings. Peripheral vasodilation
Awad (1973) examined biopsy tissues fromrestores local pH to normal, increases oxygenation
trigger points using an electron microscope andof the tissues in the area of the trigger point, and
detected a significant increase in the number ofgradually eliminates the trigger point.
platelets, which caused the release of serotoninReferences
and mast cells, which in turn released histamine.1. Awad, E.A.: Interstitial myofibrositis: hypothesis
Both serotonin and histamine are potentof the mechanism, Arch. Phys. Med. Rehab,
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body is trying to fight the local ischemia in the2. Fassbender H.G. Pathology of the Rheumatic
trigger point area. In his now classical work,Diseases. Springer-Verlag, New York, 1975
Fassbender (1975) conducted a histological3. Froriep, F. Ein Beitrag zur Pathologie und
examination of the circulation in the area of theTherapie des Rheumatismus. Weimar, 1843.
trigger point and proved once and for all that "...4. Glezer, O., Dalicho, V.A. Segmentmassage.
the trigger point represents a region of localLeipzig, 1955
ischemia." The same results were obtained by5. Glogowski, G., Wallraff, J. "Ein beitrag zur Klinik
Popelansky et al., (1986) who used radioisotopeund Histologie der Muskkelharten (Myogelosen)", Z.
evaluation of blood circulation in the area of theOrthop., 80:237-268, 1951
trigger point.6. Gogoleva, E.F. "New Approaches to Diagnosis
The End Plate Theoryand Therapy of Fibromyalgia associated with
The end plate theory is not a new theory. TravellSpondylosis." Ther. Arch., 4:40-45, 2001.
and Simmons constantly emphasize the nervous7. Heine, H. Lehbruh der biologischen Medicine.
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end-plate zones. They even name special typespoints show spontaneous needle EMG activity",
of trigger points called “motor triggerSpine, 18:1803-1807, 1993.
points,” which are located in the middle of the9. Korr, I.M. "The Neural Basis of the Osteopathic
muscle belly at the neuromuscular junction. "TheLesion." JAOA, 47(4): 191-198, 1947.
functionally significant structure with regard to the10. Kreymer, A.Y. Vibration Massage in Diseases
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junction (end-plate zone)..." and "Some triggerTomsk University, Tomsk, 1987.
points are closely associated with myoneural11. Mackenzie, J. Angina Pectoris. Henry, Frowde &
junctions, others not." (Travell and Simmons,Hodder & Stroughton, London, 1923.
1983). The idea of the nervous system and the12. Mezlack, R., Wall, P. “Pain Mechanism: A
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early as 1947 Prof. Korr addressed the same1965.
issues in his research.13. Miehlke, K., Schulze, G., Eger, W. " Klinische und
According to histological studies (Heine, 1997;experimentelle Untersuchungen zum
Gogoleva, 2001) chronic pain and low gradeFibrositis-syndrom. Z. Rheumaforsch, 19:310-330,
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responsible for the low grade inflammation around14. Popelansky, Y.Y., Zaslavsky, E.S., Veselovsky,
the terminal parts of the sensory and motorV.P. Medicosocial significance, etiology,
neurons which end in the soft tissues. This chronicpathogenesis, and diagnosis of non-articular
inflammation activates the local fibroblasts, whichdiseases of soft tissues of the lims and back.
deposit collagen around the nerve endings formingVorpr. Rheumat., 3:38-43, 1986.
so-called "collagen cuffs.” This additional15. Schade, H. "Untersuchungen in der
irritating factor triggers an afferent sensory flowErkaltungstrade: III. Uber den Rheumatismus, in
to the central nervous system, which isbesondere den Muskelrheumatismus (Myogelose)."
interpreted by the brain as pain. This mechanismMunch. Med. Wschr., 68, 95-99, 1921.
is partially described by the generation of pain in16. Travell, J.G., Simons, D.G. Myofascial Pain and
the area of motor trigger points. We have toDysfunction. The Trigger Points Manual. Williams &
consider that the terminal parts of the sensoryWilkins, Baltimore, 1983.
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